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Items 1 to 10 of about 39
1. Lawrence DW, Stewart GW: Evaluation of a program of university placement for spinal cord injured rehabilitation clients in Louisiana. J La State Med Soc; 1997 Sep;149(9):331-3
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Evaluation of a program of university placement for spinal cord injured rehabilitation clients in Louisiana.
  • The Louisiana State Department of Social Services, Rehabilitation Services (LRS) offers a program of financial support for spinal cord injured persons to attend college.
  • We evaluated this program's effectiveness and attempted to identify factors that may predict student success.
  • We examined the LRS case records of all students with documented SCI who had enrolled in the LRS college program before 1988.
  • We compared the proportion of LRS clients who graduated with the proportion of general population students attending the same schools.
  • We measured the association between graduation and factors that may help predict success.
  • Of the 51 LRS clients studied, 25 (49%) graduated from baccalaureate institutions within 6 years.
  • Of Louisiana students-at-large who attended the colleges that housed LRS students, 36% of those graduated within 6 years.
  • The best predictor of success was the American College Test score.
  • All students with a score of 17 or more graduated.
  • A larger proportion of LRS-sponsored students graduated than did students from the general population.
  • [MeSH-major] Program Evaluation. Spinal Cord Injuries / rehabilitation. Universities
  • [MeSH-minor] Adult. Educational Measurement. Humans. Louisiana. Retrospective Studies

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  • (PMID = 9316350.001).
  • [ISSN] 0024-6921
  • [Journal-full-title] The Journal of the Louisiana State Medical Society : official organ of the Louisiana State Medical Society
  • [ISO-abbreviation] J La State Med Soc
  • [Language] eng
  • [Grant] United States / PHS HHS / / U59 CCU603362
  • [Publication-type] Journal Article; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] UNITED STATES
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2. Lawrence DW, Sharma B: A review of the neuroprotective role of vitamin D in traumatic brain injury with implications for supplementation post-concussion. Brain Inj; 2016;30(8):960-8
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  • [Title] A review of the neuroprotective role of vitamin D in traumatic brain injury with implications for supplementation post-concussion.
  • BACKGROUND: Nutritional interventions are promising treatment adjuncts in the management of concussion.
  • Vitamin D (VDH) supplementation has demonstrated neuroprotective properties in multiple models of acquired brain injury.
  • OBJECTIVE: Review the neuroprotective role of VDH supplementation following traumatic brain injury (TBI).
  • METHODS: A Medline search was conducted to review manuscripts investigating the influence of VDH status or supplementation on TBI outcomes.
  • RESULTS: The search identified 165 studies, of which five were included.
  • Four manuscripts studied a rodent model of TBI, while one studied a clinical sample.
  • Vitamin D monotherapy independently reduced inflammation and neuronal injury following TBI, with a more robust effect observed in combination with progesterone (PROG).
  • One study demonstrated VDH deficiency exacerbates post-TBI inflammatory response.
  • One study in a clinical sample found combination therapy superior to PROG alone or placebo in improving outcomes after severe TBI.
  • One study observed a more robust response to low-dose VDH compared to high-dose VDH when given in combination with PROG.
  • CONCLUSION: A protective role for VDH and a vitamin D sufficient status was identified for numerous outcomes following TBI.
  • However, VDH supplementation cannot be recommended at this time to improve outcomes following TBI.

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  • (PMID = 27185224.001).
  • [ISSN] 1362-301X
  • [Journal-full-title] Brain injury
  • [ISO-abbreviation] Brain Inj
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Keywords] NOTNLM ; Concussion / neuroprotection / traumatic brain injury (TBI) / vitamin D
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3. Bruyninckx WJ, Comerford KM, Lawrence DW, Colgan SP: Phosphoinositide 3-kinase modulation of beta(3)-integrin represents an endogenous "braking" mechanism during neutrophil transmatrix migration. Blood; 2001 May 15;97(10):3251-8
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  • [Title] Phosphoinositide 3-kinase modulation of beta(3)-integrin represents an endogenous "braking" mechanism during neutrophil transmatrix migration.
  • During episodes of inflammation, neutrophils (polymorphonuclear leukocytes [PMNs]) encounter subendothelial matrix substrates that may require additional signaling pathways as directives for movement through the extracellular space.
  • Using an in vitro endothelial and epithelial model, inhibitors of phosphoinositide 3-kinase (PI3K) were observed to promote chemoattractant-stimulated migration by as much as 8 +/- 0.3-fold.
  • Subsequent studies indicated that PMNs respond in a similar manner to RGD-containing matrix substrates and that PMN-matrix interactions are potently inhibited by antibodies directed against beta(3)- but not beta(1)-integrin antibodies, and that PI3K inhibitors block beta(3)-integrin dependence.
  • Biochemical analysis of intracellular beta(3)-integrin uncoupling by PI3K inhibitors revealed diminished beta(3)-integrin tyrosine phosphorylation and decreased association with p72(syk).
  • Similarly, the p72(syk) inhibitor piceatannol promoted PMN transmatrix migration, whereas HIV-tat peptide-facilitated loading of peptides corresponding to the beta(3)-integrin cytoplasmic tail identified the functional tyrosine residues for this activity.
  • These data indicate that PI3K-regulated beta(3)-integrin represents a natural "braking" mechanism for PMNs during transit through the extracellular matrix.
  • [MeSH-major] Antigens, CD / physiology. Chemotaxis, Leukocyte. Extracellular Matrix / physiology. Neutrophils / physiology. Phosphatidylinositol 3-Kinases / metabolism. Platelet Membrane Glycoproteins / physiology
  • [MeSH-minor] Androstadienes / pharmacology. Antibodies, Monoclonal / pharmacology. Cell Line. Cytoplasm / chemistry. Endothelium, Vascular / physiology. Enzyme Inhibitors / pharmacology. Enzyme Precursors / antagonists & inhibitors. Enzyme Precursors / physiology. Epithelium / physiology. Humans. Integrin beta3. Intracellular Signaling Peptides and Proteins. Leukotriene B4 / pharmacology. N-Formylmethionine Leucyl-Phenylalanine / pharmacology. Peroxidase / analysis. Phosphotyrosine / metabolism. Protein-Tyrosine Kinases / antagonists & inhibitors. Protein-Tyrosine Kinases / physiology

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  • (PMID = 11342456.001).
  • [ISSN] 0006-4971
  • [Journal-full-title] Blood
  • [ISO-abbreviation] Blood
  • [Language] eng
  • [Grant] United States / NIDDK NIH HHS / DK / DK50189; United States / NHLBI NIH HHS / HL / HL60569; United States / NIDCR NIH HHS / DE / P0-1 DE13499
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Androstadienes; 0 / Antibodies, Monoclonal; 0 / Antigens, CD; 0 / Enzyme Inhibitors; 0 / Enzyme Precursors; 0 / Integrin beta3; 0 / Intracellular Signaling Peptides and Proteins; 0 / Platelet Membrane Glycoproteins; 1HGW4DR56D / Leukotriene B4; 21820-51-9 / Phosphotyrosine; 59880-97-6 / N-Formylmethionine Leucyl-Phenylalanine; EC 1.11.1.7 / Peroxidase; EC 2.7.1.- / Phosphatidylinositol 3-Kinases; EC 2.7.10.1 / Protein-Tyrosine Kinases; EC 2.7.10.1 / Syk kinase; XVA4O219QW / wortmannin
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4. Lawrence DW, Stewart GW, Christy DM, Gibbs LI, Ouellette M: High school football-related cervical spinal cord injuries in Louisiana: the athlete's perspective. J La State Med Soc; 1997 Jan;149(1):27-31
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  • [Title] High school football-related cervical spinal cord injuries in Louisiana: the athlete's perspective.
  • Louisiana has one of the highest rates in the nation of cervical spinal cord injuries to high school football players.
  • When the national rate of these injuries is applied to the number of high school participants in Louisiana, we would expect there to be only one catastrophic neck injury every 14 years.
  • Louisiana, however, has averaged 2.3 spinal cord injuries per year for the past seven football seasons.
  • Players who use the top of their helmets to tackle, block, or strike opponents are at greatest risk for these injuries.
  • This study was undertaken to describe the safe tackling knowledge, attitudes, and practices of Louisiana high school football players.
  • We surveyed 596 players from 16 Louisiana high schools.
  • When asked if it was within the rules to tackle anyone by using the top of their helmet, 29% incorrectly answered "yes".
  • Similarly, when asked if they had ever tackled anyone using the top of their helmet, 33% reported that they had.
  • Twenty-eight percent said that they had been taught to use this unsafe method.
  • Of these, 83% said that their coach taught them this dangerous and illegal method.
  • Using the helmet as a battering ram must be discouraged.
  • Education for officials, coaches, and players is needed to improve recognition of improper tackling.
  • Proper training in tackling and blocking is an important means of minimizing the possibility of catastrophic injury.
  • [MeSH-major] Football / injuries. Health Knowledge, Attitudes, Practice. Spinal Cord Injuries / prevention & control
  • [MeSH-minor] Adolescent. Adult. Cross-Sectional Studies. Head Protective Devices. Health Education. Humans. Incidence. Louisiana / epidemiology. Population Surveillance

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  • (PMID = 9033192.001).
  • [ISSN] 0024-6921
  • [Journal-full-title] The Journal of the Louisiana State Medical Society : official organ of the Louisiana State Medical Society
  • [ISO-abbreviation] J La State Med Soc
  • [Language] eng
  • [Grant] United States / PHS HHS / / U59 CCU603362
  • [Publication-type] Journal Article; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] UNITED STATES
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5. Lawrence DW, Koenig JM: Enhanced phagocytosis in neonatal monocyte-derived macrophages is associated with impaired SHP-1 signaling. Immunol Invest; 2012;41(2):129-43
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  • [Title] Enhanced phagocytosis in neonatal monocyte-derived macrophages is associated with impaired SHP-1 signaling.
  • Resident macrophages represent a first line of defense through the ingestion of microbial pathogens.
  • Phagocytosis mediated by immunoglobulin-binding Fc receptors is a complex series of events involving tyrosine phosphorylation and dephosphorylation.
  • In the present study we determined that the phagocytic capacity of neonatal monocyte-derived macrophage (MDM) was enhanced in comparison to adult MDM.
  • Cross-linking of surface FcγRIIa receptors enhanced tyrosine phosphorylation of several proteins in both groups, followed by a reduction in tyrosine phosphorylation in adult but not neonatal MDM.
  • Expression of the tyrosine phosphatase SHP-1 was similar in neonatal and adult MDM; however, baseline SHP-1 activity levels were diminished in neonatal MDM.
  • Cross-linking of FcγRIIa receptors induced a greater increase in SHP-1 activity in adult MDM vs. neonatal MDM.
  • The cytoplasmic adapter protein Cbl is a SHP-1 ligand and negatively affects phagocytosis.
  • As determined by co-immunoprecipitation assays, SHP-1 and Cbl did not associate to the same extent in neonatal as compared to adult MDM.
  • Our data suggest that the enhanced phagocytic capacity of neonatal MDM is associated with decreased SHP-1 activity and alteration of downstream signaling pathways.
  • [MeSH-major] Macrophages / immunology. Phagocytosis. Protein Tyrosine Phosphatase, Non-Receptor Type 6 / metabolism. Proto-Oncogene Proteins c-cbl / metabolism. Receptors, IgG / metabolism
  • [MeSH-minor] Adult. Cells, Cultured. Enzyme Activation. Humans. Infant, Newborn. Phosphorylation. Protein Transport. Receptor Aggregation. Signal Transduction

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  • (PMID = 21806449.001).
  • [ISSN] 1532-4311
  • [Journal-full-title] Immunological investigations
  • [ISO-abbreviation] Immunol. Invest.
  • [Language] eng
  • [Grant] United States / NICHD NIH HHS / HD / HD47401
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / FCGR2A protein, human; 0 / Receptors, IgG; EC 3.1.3.48 / Protein Tyrosine Phosphatase, Non-Receptor Type 6; EC 6.3.2.- / CBL protein, human; EC 6.3.2.- / Proto-Oncogene Proteins c-cbl
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6. Lawrence DW, Blackledge VO: Protocol for evaluation of the effect of hearing aid electroacoustic parameters on perception of amplified speech. J Am Audiol Soc; 1977 May-Jun;2(6):197-201
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  • [Title] Protocol for evaluation of the effect of hearing aid electroacoustic parameters on perception of amplified speech.
  • A protocol for the evaluation of the effects of changes in hearing aid electroacoustic parameters was developed and evaluated.
  • The protocol called for the creation of a matrix with as many dimensions as there are parameters to be evaluated.
  • The protocol also called for each dimension to have as many divisions as there are possible variations of a parameter.
  • The cell entries in the matrix were the listener speech discrimination scores in noise.
  • It was found that one cell within the matrix always uniquely contained the highest listener speech discrimination score.
  • It was concluded that use of such a protocol would allow the establishment of the precise electroacoustic settings of a master hearing aid which would result in best speech understanding for the listener.
  • [MeSH-major] Auditory Perception. Hearing Aids. Speech
  • [MeSH-minor] Audiometry / instrumentation. Auditory Threshold. Correction of Hearing Impairment. Female. Humans. Male. Middle Aged

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  • (PMID = 893188.001).
  • [ISSN] 0360-9294
  • [Journal-full-title] Journal of the American Audiology Society
  • [ISO-abbreviation] J Am Audiol Soc
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] UNITED STATES
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7. Lawrence DW, Pryzwansky KB: The vasodilator-stimulated phosphoprotein is regulated by cyclic GMP-dependent protein kinase during neutrophil spreading. J Immunol; 2001 May 1;166(9):5550-6
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  • [Title] The vasodilator-stimulated phosphoprotein is regulated by cyclic GMP-dependent protein kinase during neutrophil spreading.
  • The expression and phosphorylation state of the vasodilator-stimulated phosphoprotein (VASP), a membrane-associated focal adhesion protein, was investigated in human neutrophils.
  • Adhesion and spreading of neutrophils induced the rapid phosphorylation of VASP.
  • The phosphorylation of VASP was dependent on cell spreading, as VASP was expressed as a dephosphorylated protein in round adherent cells and was phosphorylated at the onset of changes in cell shape from round to spread cells.
  • Immunofluorescence microscopy demonstrated that VASP was localized at the cell cortex in round cells and redistributed to focal adhesions at the ventral surface of the cell body during cell spreading.
  • Dual labeling of spread cells indicated that VASP was colocalized with F-actin in filopodia and in focal adhesions, suggesting that the phosphorylation of VASP during cell spreading may be involved in focal adhesion complex organization and actin dynamics.
  • VASP is a prominent substrate for both cGMP-dependent protein kinase (cGK) and cAMP-dependent protein kinase.
  • Evidence suggested that cGK regulated neutrophil spreading, as both VASP phosphorylation and neutrophil spreading were inhibited by Rp-8-pCPT-cGMPS (cGK inhibitor), but not KT5720 (cAMP-dependent protein kinase inhibitor).
  • In contrast, neutrophil spreading was accelerated when cGMP levels were elevated with 8-Br-cGMP, a direct activator of cGK.
  • Furthermore, the same conditions that lead to VASP phosphorylation during neutrophil adherence and spreading induced significant elevations of cGMP in neutrophils.
  • These results indicate that cGMP/cGK signal transduction is required for neutrophil spreading, and that VASP is a target for cGK regulation.
  • [MeSH-major] Cell Adhesion Molecules / metabolism. Cell Size / physiology. Cyclic GMP-Dependent Protein Kinases / physiology. Neutrophils / cytology. Neutrophils / metabolism. Phosphoproteins / metabolism
  • [MeSH-minor] Cell Adhesion / physiology. Cyclic GMP / metabolism. Humans. Microfilament Proteins. Microscopy, Fluorescence. Phosphorylation. Second Messenger Systems / physiology

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  • (PMID = 11313394.001).
  • [ISSN] 0022-1767
  • [Journal-full-title] Journal of immunology (Baltimore, Md. : 1950)
  • [ISO-abbreviation] J. Immunol.
  • [Language] eng
  • [Grant] United States / NIEHS NIH HHS / ES / ES07017; United States / NIGMS NIH HHS / GM / GM55336-02
  • [Publication-type] Journal Article; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Cell Adhesion Molecules; 0 / Microfilament Proteins; 0 / Phosphoproteins; 0 / vasodilator-stimulated phosphoprotein; EC 2.7.11.12 / Cyclic GMP-Dependent Protein Kinases; H2D2X058MU / Cyclic GMP
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8. Lawrence DW, Hutchison M: Concerns with novel concussion protocol. Br J Sports Med; 2017 Apr;51(7):620-621
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  • [Title] Concerns with novel concussion protocol.

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  • (PMID = 27872042.001).
  • [ISSN] 1473-0480
  • [Journal-full-title] British journal of sports medicine
  • [ISO-abbreviation] Br J Sports Med
  • [Language] eng
  • [Publication-type] Letter
  • [Publication-country] England
  • [Keywords] NOTNLM ; Concussion
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9. Lawrence DW, Bruyninckx WJ, Louis NA, Lublin DM, Stahl GL, Parkos CA, Colgan SP: Antiadhesive role of apical decay-accelerating factor (CD55) in human neutrophil transmigration across mucosal epithelia. J Exp Med; 2003 Oct 6;198(7):999-1010
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  • [Title] Antiadhesive role of apical decay-accelerating factor (CD55) in human neutrophil transmigration across mucosal epithelia.
  • Neutrophil migration across mucosal epithelium during inflammatory episodes involves the precise orchestration of a number a cell surface molecules and signaling pathways.
  • After successful migration to the apical epithelial surface, apically localized epithelial proteins may serve to retain PMN at the lumenal surface.
  • At present, identification of apical epithelial ligands and their PMN counter-receptors remain elusive.
  • Therefore, to define the existence of apical epithelial cell surface proteins involved in PMN-epithelial interactions, we screened a panel of antibodies directed against epithelial plasma membranes.
  • This strategy identified one antibody (OE-1) that both localized to the apical cell membrane and significantly inhibited PMN transmigration across epithelial monolayers.
  • Microsequence analysis revealed that OE-1 recognized human decay-accelerating factor (DAF, CD55).
  • DAF is a highly glycosylated, 70-80-kD, glycosyl-phosphatidyinositol-linked protein that functions predominantly as an inhibitor of autologous complement lysis.
  • DAF suppression experiments using antisense oligonucleotides or RNA interference revealed that DAF may function as an antiadhesive molecule promoting the release of PMN from the lumenal surface after transmigration.
  • Similarly, peptides corresponding to the antigen recognition domain of OE-1 resulted in accumulation of PMN on the apical epithelial surface.
  • The elucidation of DAF as an apical epithelial ligand for PMN provides a target for novel anti-inflammatory therapies directed at quelling unwanted inflammatory episodes.

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  • (PMID = 14530374.001).
  • [ISSN] 0022-1007
  • [Journal-full-title] The Journal of experimental medicine
  • [ISO-abbreviation] J. Exp. Med.
  • [Language] ENG
  • [Grant] United States / NHLBI NIH HHS / HL / R01 HL054229; United States / NIDDK NIH HHS / DK / DK 50189; United States / NIDDK NIH HHS / DK / R01 DK050189; United States / NHLBI NIH HHS / HL / HL 54229; United States / NIDCR NIH HHS / DE / P01 DE013499; United States / NIDCR NIH HHS / DE / DE 13499; United States / NIDDK NIH HHS / DK / R29 DK050189; United States / NIDDK NIH HHS / DK / R37 DK050189
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antigens, CD55
  • [Other-IDs] NLM/ PMC2194216
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10. Lawrence DW: Re: "Cancer incidence near radio and television transmitters in Great Britain". Am J Epidemiol; 1997 Oct 15;146(8):682-3
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Re: "Cancer incidence near radio and television transmitters in Great Britain".
  • [MeSH-major] Environmental Exposure / adverse effects. Neoplasms / epidemiology. Radio Waves / adverse effects
  • [MeSH-minor] Great Britain / epidemiology. Humans. Incidence. Television

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  • [CommentOn] Am J Epidemiol. 1997 Jan 1;145(1):1-9 [8982016.001]
  • [CommentOn] Am J Epidemiol. 1997 Jan 1;145(1):10-7 [8982017.001]
  • (PMID = 9345124.001).
  • [ISSN] 0002-9262
  • [Journal-full-title] American journal of epidemiology
  • [ISO-abbreviation] Am. J. Epidemiol.
  • [Language] eng
  • [Publication-type] Comment; Letter
  • [Publication-country] UNITED STATES
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